October 24, 2011

笨豬pppp只是井底之蛙 , 我引用的資料都是最近幾年表觀遺傳學的研究, 大白癡pppp用20世紀末的遺傳學來反駁, 那麼再多的辯論也是白費口舌

gfhfgdhj於<cite>2011/10/24 21:30 </cite>回應

-----------臺灣大學小兒遺傳學教授胡務亮,引[表觀遺傳學] 和長庚大學婦產科教授 徐振傑引用[孟德爾遺傳學] ,在建中校友會也辯論過 [前夫遺傳]的可能性  --前夫的DNA密碼可能對胚胎基因的修飾,但並不會造成基因序列本身的改變。基因外的修飾包括如分子的增加,像是增加甲基群到基因的骨架上。增加的這些分子將改變基因的結構與外觀,因而影響基因在細胞核中與重要轉錄因子之交互作用'這類嶄新的研究正在改變整個遺傳學領域甚至發展成為一個副學門,叫(epigenetics)表觀遺傳學又稱「擬遺傳學」、「表遺傳學」、「外遺傳學」以及「後遺傳學」

gfhfgdhj於<cite>2011/10/24 21:27 </cite>回應

utuytyty於<cite>2011/10/24 18:13 </cite>回應

 

pppp你是笨豬王八'才是在浪費時間'快四十歲 沒有搞過真處女 '吃不到葡萄說葡萄酸,只會網路貼文章''有本事,你就釣個真處女

gfhfgdhj於<cite>2011/10/24 21:26 </cite>回應

笨豬PPPP邏輯錯誤'真是丟人現眼,無恥阿。 

gfhfgdhj於<cite>2011/10/24 21:24 </cite>回應

笨豬pppp只是井底之蛙 , 我引用的資料都是最近幾年表觀遺傳學的研究, 笨蛋pppp用20世紀末的遺傳學來反駁, 那麼再多的辯論也是白費口舌 

gfhfgdhj於<cite>2011/10/24 21:22 </cite>回應

笨豬pppp只是井底之蛙

 
 
莊子·秋水》一開始講述了河伯和北海的對話,秋天黃河漲水,河面陡然變寬,寬到連對岸的都無法看清。河伯洋洋自得,以為自己很了不起,但當其來到入海口看到大海之時,才知道自己何等渺小。北海說道:「井蛙不可以語于海者,拘於虛也」。 本篇其後又講述了井蛙的完整故事,說得是一隻青蛙住在一個陷井[1] 之中,一天遇到了一隻來自東海的巨。青蛙對鱉說:「你看我多快樂啊,出去可以在井邊跳來跳去,回來了可以在井裡的中休息,在水中可以只把嘴巴露出來,還可以把踩在軟軟的里。那些蝦米螃蟹蝌蚪哪個能比得上我?我就是這一井之主,多麼地快樂!你幹嘛不常到井裡來看看呢?」那隻巨鱉於是想進到井裡,可井卻小得連腳都放不進去。鱉對青蛙說:「你見過大海嗎?其闊何止千里?其深何止千大禹治水之時十年中有九年洪水,也沒有使大海水量增加,商湯之時八年中有七年旱災,海水也沒有減少多少。大海如此不受洪水大旱的影響,這才是住在東海的大快樂呢。」井裡的青蛙聽了,驚呆了,方才知道自己所居之地是何等的微不足道。 這則寓言深刻諷刺了沒有見過大世面的人眼界狹小。

gfhfgdhj於<cite>2011/10/24 21:18 </cite>回應

笨豬pppp只是井底之蛙 , 我引用的資料都是最近幾年表觀遺傳學的研究, 如果有笨蛋用20世紀末的遺傳學來反駁, 那麼再多的辯論也是白費口舌 
, 就像子宮頸癌的原因-- 1999 和2011的課本也是不同原因 ---------------臺灣大學小兒遺傳學教授胡務亮,引[表觀遺傳學] 和長庚大學婦產科教授 徐振傑引用[孟德爾遺傳學] ,在建中校友會也辯論過 [前夫遺傳]的可能性  --前夫的DNA密碼可能對胚胎基因的修飾,但並不會造成基因序列本身的改變。基因外的修飾包括如分子的增加,像是增加甲基群到基因的骨架上。增加的這些分子將改變基因的結構與外觀,因而影響基因在細胞核中與重要轉錄因子之交互作用'這類嶄新的研究正在改變整個遺傳學領域甚至發展成為一個副學門,叫(epigenetics)表觀遺傳學又稱「擬遺傳學」、「表遺傳學」、「外遺傳學」以及「後遺傳學」(英文epigenetics),是一門生物學學科,研究在沒有細胞核DNA序列改變的情況時,基因功能的可逆的、可遺傳的改變。這些改變包括DNA的修飾(如甲基化修飾)、組蛋白的各種修飾等。也指生物發育過程中包含的程序的研究。在這兩種情況下,研究的對象都包括在DNA序列中未包含的基因調控信息如何傳遞到(細胞生物體的)下一代這個問題。在西方文字中epigenetics即指在DNA包含的遺傳信息以外附加的希臘文前綴epi-)。

表觀遺傳學是逐漸興起的一門學科,是在研究與經典孟德爾遺傳學遺傳法則不相符的許多生命現象過程中逐步發展起來的。

表觀遺傳現象包括DNA甲基化RNA干擾組蛋白修飾等。與經典遺傳學以研究基因序列影響生物學功能為核心相比,表觀遺傳學主要研究這些「表觀遺傳現象」的建立和維持的機制。


而以表觀遺傳學來看,完蛋的不是《處女情節的科學依據》一文   所以[前夫效應]不是胡說八道。謠言止於智者。笨豬pppp   才是胡說八道瞎扯

gfhfgdhj於<cite>2011/10/24 20:47 </cite>回應

笨蛋pppp 應該看不懂以下的遺傳學論文----The approach to genetics investigated in this paper is unorthodox-modifications acquired somatically during the lifetime
of an animal are not expected to undergo hereditary transmission
(Weismann's doctrine, 5). We have shown, however,
that a neonatally acquired and actively maintained state of
antigen-specific tolerance to foreign H-2 antigens in male CBA
mice is transmitted to a high proportion (50-60%) of firstgeneration
offspring. All 10 of the A/J tolerant males produced
such progeny [although there was some variation among the
fathers in the efficiency of transmission (Fig. 1A)]. Further
breeding (incrossing and outcrossing) from first-generation mice
showed that a high proportion (;50%) of second-generation
animals were also specifically tolerant or hyporesponders to the
original antigen. This specific tolerant state was transmitted in
the absence of further exposure to F1 cells and presumably
represents the relatively stable inheritance of a trait derived
from the original parental CBA male. More recent studies have
demonstrated a similar transmission of tolerance (to B10.D2 or
B1O.BR antigens) in B10 male animals exposed neonatally to
foreign lymphocytes (unpublished data).

 
If a genetic mechanism is responsible for the phenomenon
observed, it requires a process whereby somatic genes (normal
or mutated) enter the germ line. A hypothetical scheme based
on two current biological theories has been discussed elsewhere
(5). (i) The clonal selection and somatic mutation theory of
antibody (idiotype) diversity [see Burnet (12) and others
(13-19)]. (ii) Temin's protovirus and provirus hypotheses on
the origin and biological significance of vertebrate type C RNA
tumor viruses (20-22). In this scheme, clonal expression and
somatic selection of a given gene copy (e.g., RNA sequences)
enhances the probability of its capture by endogenous RNA
virus particles, leading ultimately to integration of somatic RNA
gene copies (via reverse transcriptase) into germ-line DNA.
 
Two additional points need consideration. First, what is the
nature of the "message" transmitted by tolerant male animals?
This could be genetic information involved in the active regulation
of tolerance in these males (e.g., see refs. 7-9) or genes
coding for expression of the MHC antigens (H-2Dd) of the F1
cells coexisting within these animals [perhaps as seen earlier by
Kanazawa and Imai (23)]. Second, of 219 first-generation mice
tested, 44% were tolerant and 29% were hyporesponders to A/J
antigens; in the second generation (176 mice), the corresponding
values were 13 and 40%. Perhaps the "germ-line" stability of
transmitted information depends upon continued exposure to
the inductive stimulus.
Irrespective of the answer to these questions, we believe that
the data document that a specifically acquired somatic characteristic
(tolerance to MHC antigens) can be transmitted, via
male mice, to subsequent generations at high frequency.
 

gfhfgdhj於<cite>2011/10/24 20:43 </cite>回應

笨豬pppp   有無   知識----------------------------20世紀80年代初,加拿大安大略癌症研究所的兩位免疫學家Gorczynski R M和鋼E J在《美國國家科學院報》上提出,小鼠通過注射骨髓細胞,、淋巴細胞,可後天獲得抗原耐受性,並能將此耐受性傳給下一代。該論文掀起了拉馬克主義在免疫學中復活了的小浪潮,英國《自然》上也為此發表評論文章和後繼論文

gfhfgdhj於<cite>2011/10/24 20:42 </cite>回應

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